IDM
Institut d' investigació
New York University
Nueva York, Estados UnidosPublicaciones en colaboración con investigadores/as de New York University (10)
2015
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Cyclometalated Iminophosphorane Gold(III) and Platinum(II) Complexes. A Highly Permeable Cationic Platinum(II) Compound with Promising Anticancer Properties
Journal of Medicinal Chemistry, Vol. 58, Núm. 15, pp. 5825-5841
2010
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Stimulation of wound revascularization by adenosine receptor activation
Extracellular ATP and Adenosine as Regulators of Endothelial Cell Function: Implications for Health and Disease (Springer Netherlands), pp. 95-112
2007
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The antiinflammatory mechanism of methotrexate depends on extracellular conversion of adenine nucleotides to adenosine by ecto-5′-nucleotidase: Findings in a study of ecto-5′-nucleotidase gene-deficient mice
Arthritis and Rheumatism, Vol. 56, Núm. 5, pp. 1440-1445
2005
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An interaction between genetic factors and gender determines the magnitude of the inflammatory response in the mouse air pouch model of acute inflammation
Inflammation, Vol. 29, Núm. 1, pp. 1-7
2002
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Adenosine promotes wound healing and mediates angiogenesis in response to tissue injury via occupancy of A2A receptors
American Journal of Pathology, Vol. 160, Núm. 6, pp. 2009-2018
2001
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Anti-inflammatory levels of aspirin and salicylate inhibit integrindependent human neutrophil aggregation and PMN influx in COX 2-and NFkB (P105) - Knockout mice
ADVANCES IN PROSTAGLANDIN AND LEUKOTRIENE RESEARCH: BASIC SCIENCE AND NEW CLINICAL APPLICATIONS
1999
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Salicylates and sulfasalazine, but not glucocorticoids, inhibit leukocyte accumulation by an adenosine-dependent mechanism that is independent of inhibition of prostaglandin synthesis and p105 of NFκB
Proceedings of the National Academy of Sciences of the United States of America, Vol. 96, Núm. 11, pp. 6377-6381
1997
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Wound healing is accelerated by agonists of adenosine A2 (G(αs)- linked) receptors
Journal of Experimental Medicine, Vol. 186, Núm. 9, pp. 1615-1620
1996
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Adenosine A2 receptor occupancy regulates stimulated neutrophil function via activation of a serine/threonine protein phosphatase
Journal of Biological Chemistry, Vol. 271, Núm. 29, pp. 17114-17118
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The anti-inflammatory mechanism of sulfasalazine is related to adenosine release at inflamed sites
Journal of Immunology, Vol. 156, Núm. 5, pp. 1937-1941