JOSE LUIS
GARCIA GIMENEZ
AYUDANTE DOCTOR/A
Instituto de Investigación Sanitaria La Fe
Valencia, EspañaPublicaciones en colaboración con investigadores/as de Instituto de Investigación Sanitaria La Fe (11)
2024
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Role of miRNAs as epigenetic regulators of immune checkpoints in lung cancer immunity
International Review of Cell and Molecular Biology
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miRNAs Related to Immune Checkpoint Inhibitor Response: A Systematic Review
International Journal of Molecular Sciences, Vol. 25, Núm. 3
2023
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Age-Related microRNA Overexpression in Lafora Disease Male Mice Provides Links between Neuroinflammation and Oxidative Stress
International Journal of Molecular Sciences, Vol. 24, Núm. 2
2022
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MicroRNA-185-5p: a marker of brain-sparing in foetuses with late-onset growth restriction
Epigenetics, Vol. 17, Núm. 11, pp. 1345-1356
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Overexpression of microRNAs miR-25-3p, miR-185-5p and miR-132-3p in late onset fetal growth restriction, validation of results and study of the biochemical pathways involved
International Journal of Molecular Sciences, Vol. 23, Núm. 1
2021
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DNA Methylation Analysis to Unravel Altered Genetic Pathways Underlying Early Onset and Late Onset Neonatal Sepsis. A Pilot Study
Frontiers in Immunology, Vol. 12
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Epigenetics in spine curvature disorders
Epigenetics in Precision Medicine (Elsevier), pp. 449-469
2020
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MicroRNA-148b-3p and MicroRNA-25-3p Are Overexpressed in Fetuses with Late-Onset Fetal Growth Restriction
Fetal Diagnosis and Therapy, Vol. 47, Núm. 9, pp. 665-674
2019
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Corrigendum to “A Drosophila model of GDAP1 function reveals the involvement of insulin signalling in the mitochondria-dependent neuromuscular degeneration” [Biochim. Biophys. Acta 1863 (2017) 801–809](S0925443917300078)(10.1016/j.bbadis.2017.01.003)
Biochimica et Biophysica Acta - Molecular Basis of Disease
2017
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A Drosophila model of GDAP1 function reveals the involvement of insulin signalling in the mitochondria-dependent neuromuscular degeneration
Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1863, Núm. 3, pp. 801-809
2014
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Oxidative stress and mitochondrial dysfunction in Kindler syndrome
Orphanet journal of rare diseases, Vol. 9, pp. 211