Publicaciones en las que colabora con JOSE M CENTENO GUIL (20)

2019

  1. Potassium channels contribute to the increased sensitivity of the rabbit carotid artery to hydrogen sulfide in diabetes

    European Journal of Pharmacology, Vol. 853, pp. 33-40

2018

  1. Diabetes modifies the role of prostanoids and potassium channels which regulate the hypereactivity of the rabbit renal artery to BNP

    Naunyn-Schmiedeberg's Archives of Pharmacology, Vol. 391, Núm. 5, pp. 501-511

  2. Molecular mechanisms underlying the neuroprotective role of atrial natriuretic peptide in experimental acute ischemic stroke

    Molecular and Cellular Endocrinology, Vol. 472, pp. 1-9

2017

  1. Mechanisms involved in the increased sensitivity of the rabbit basilar artery to atrial natriuretic peptide in diabetes

    European Journal of Pharmacology, Vol. 815, pp. 64-72

  2. Molecular mechanisms mediating the neuroprotective role of the selective estrogen receptor modulator, bazedoxifene, in acute ischemic stroke: A comparative study with 17β-estradiol

    Journal of Steroid Biochemistry and Molecular Biology, Vol. 171, pp. 296-304

2014

  1. The selective estrogen receptor modulator, bazedoxifene, reduces ischemic brain damage in male rat

    Neuroscience Letters, Vol. 575, pp. 53-57

2013

  1. Involvement of prostacyclin and potassium channels in the diabetes-induced hyporeactivity of the rabbit carotid artery to B-type natriuretic peptide

    European Journal of Pharmacology, Vol. 701, Núm. 1-3, pp. 159-167

2012

  1. Diabetes impairs the atrial natriuretic peptide relaxant action mediated by potassium channels and prostacyclin in the rabbit renal artery

    Pharmacological Research, Vol. 66, Núm. 5, pp. 392-400

2011

  1. Mechanisms underlying the diabetes-induced hyporeactivity of the rabbit carotid artery to atrial natriuretic peptide

    Pharmacological Research, Vol. 63, Núm. 3, pp. 190-198

2010

  1. Mechanisms involved in the relaxant action of testosterone in the renal artery from male normoglycemic and diabetic rabbits

    Pharmacological Research, Vol. 61, Núm. 2, pp. 149-156

  2. Role of NO-synthases and cyclooxygenases in the hyperreactivity of male rabbit carotid artery to testosterone under experimental diabetes

    Pharmacological Research, Vol. 61, Núm. 1, pp. 62-70

2004

  1. Mechanisms underlying diabetes enhancement of endothelin-1-induced contraction in rabbit basilar artery

    European Journal of Pharmacology, Vol. 486, Núm. 3, pp. 289-296

2002

  1. Experimental diabetes induces hyperreactivity of rabbit renal artery to 5-hydroxytryptamine

    European Journal of Pharmacology, Vol. 439, Núm. 1-3, pp. 121-127

2001

  1. Diabetes potentiates acetylcholine-induced relaxation in rabbit renal arteries

    European Journal of Pharmacology, Vol. 415, Núm. 2-3, pp. 225-232

2000

  1. Diabetes-induced changes in endothelial mechanisms implicated in rabbit carotid arterial response to 5-hydroxytryptamine

    European Journal of Pharmacology, Vol. 401, Núm. 3, pp. 397-402

  2. Influence of experimental diabetes on regulatory mechanisms of vascular response of rabbit carotid artery to acetylcholine

    Life Sciences, Vol. 66, Núm. 21, pp. 2071-2080

1998

  1. Comparative analysis of the vascular actions of diterpenes isolated from Euphorbia canariensis

    Journal of Pharmacy and Pharmacology, Vol. 50, Núm. 2, pp. 237-241

1997

  1. Analysis of rabbit vascular responses to DBI, an ingol derivative isolated from Euphorbia canariensis

    Journal of Pharmacy and Pharmacology, Vol. 49, Núm. 5, pp. 573-576

  2. Analysis of the vascular effects of an ingenol derivative isolated from Euphorbia canariensis

    Pharmaceutical Sciences, Vol. 3, Núm. 2, pp. 113-116

  3. Impairment of the modulatory role of nitric oxide on the endothelin-1- elicited contraction of cerebral arteries: A pathogenetic factor in cerebral vasospasm after subarachnoid hemorrhage?

    Neurosurgery, Vol. 41, Núm. 1, pp. 245-253