Publications in collaboration with researchers from Universidad de Córdoba (9)

2019

  1. Deregulation of miR-324/KISS1/kisspeptin in early ectopic pregnancy: mechanistic findings with clinical and diagnostic implications

    American Journal of Obstetrics and Gynecology, Vol. 220, Núm. 5, pp. 480.e1-480.e17

2014

  1. Dopamine receptor 2 activation inhibits ovarian vascular endothelial growth factor secretion in an ovarian hyperstimulation syndrome (OHSS) animal model: Implications for treatment of OHSS with dopamine receptor 2 agonists

    Fertility and Sterility, Vol. 102, Núm. 5, pp. 1468-1476.e1

  2. Dopamine receptor 2 activation inhibits ovarian vascular endothelial growth factor secretion in vitro: Implications for treatment of ovarian hyperstimulation syndrome with dopamine receptor 2 agonists

    Fertility and Sterility, Vol. 101, Núm. 5

  3. The Lin28/Let-7 system in early human embryonic tissue and ectopic pregnancy

    PLoS ONE, Vol. 9, Núm. 1

2011

  1. Evidences for the existence of a low dopaminergic tone in polycystic ovarian syndrome: Implications for OHSS development and treatment

    Journal of Clinical Endocrinology and Metabolism, Vol. 96, Núm. 8, pp. 2484-2492

  2. The effects of ergot and non-ergot-derived dopamine agonists in an experimental mouse model of endometriosis

    Reproduction, Vol. 142, Núm. 5, pp. 745-755

2009

  1. KiSS-1 in the mammalian ovary: Distribution of kisspeptin in human and marmoset and alterations in KiSS-1 mRNA levels in a rat model of ovulatory dysfunction

    American Journal of Physiology - Endocrinology and Metabolism, Vol. 296, Núm. 3

2006

  1. Expression of KiSS-1 in rat ovary: Putative local regulator of ovulation?

    Endocrinology, Vol. 147, Núm. 10, pp. 4852-4862

  2. Low-dose dopamine agonist administration blocks vascular endothelial growth factor (VEGF)-mediated vascular hyperpermeability without altering VEGF receptor 2-dependent luteal angiogenesis in a rat ovarian hyperstimulation model

    Endocrinology, Vol. 147, Núm. 11, pp. 5400-5411