JUAN VICENTE
ESPLUGUES MOTA
CATEDRÁTICO/A DE UNIVERSIDAD
VICTOR MANUEL
VICTOR GONZALEZ
TITULAR DE UNIVERSIDAD
Publications dans lesquelles il/elle collabore avec VICTOR MANUEL VICTOR GONZALEZ (17)
2024
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Interference with mitochondrial function as part of the antifibrogenic effect of Rilpivirine: A step towards novel targets in hepatic stellate cell activation
Biomedicine and Pharmacotherapy, Vol. 178
2023
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Anti-inflammatory and immunomodulating effects of rilpivirine: Relevance for the therapeutics of chronic liver disease
Biomedicine and Pharmacotherapy, Vol. 167
2021
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Understanding the implication of autophagy in the activation of hepatic stellate cells in liver fibrosis: are we there yet?
Journal of Pathology, Vol. 254, Núm. 3, pp. 216-228
2019
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Mitophagy in human astrocytes treated with the antiretroviral drug Efavirenz: Lack of evidence or evidence of the lack
Antiviral Research, Vol. 168, pp. 36-50
2016
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The purine analogues abacavir and didanosine increase acetaminophen-induced hepatotoxicity by enhancing mitochondrial dysfunction
Journal of Antimicrobial Chemotherapy, Vol. 71, Núm. 4, pp. 916-926
2015
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Mitochondrial (dys)function - A factor underlying the variability of efavirenz-induced hepatotoxicity?
British Journal of Pharmacology, Vol. 172, Núm. 7, pp. 1713-1727
2013
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ER stress in human hepatic cells treated with Efavirenz: Mitochondria again
Journal of Hepatology, Vol. 59, Núm. 4, pp. 780-789
2010
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Inhibition of mitochondrial function by efavirenz increases lipid content in hepatic cells
Hepatology, Vol. 52, Núm. 1, pp. 115-125
2009
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Mitochondrial-targeted antioxidants and oxidative stress: A proteomic prospective study
Current Pharmaceutical Design, Vol. 15, Núm. 26, pp. 3052-3062
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Oxidative stress and mitochondrial dysfunction in sepsis: A potential therapy with mitochondria-targeted antioxidants
Infectious Disorders - Drug Targets, Vol. 9, Núm. 4, pp. 376-389
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Regulation of oxygen distribution in tissues by endothelial nitric oxide
Circulation Research, Vol. 104, Núm. 10, pp. 1178-1183
2008
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Endothelial nitric oxide synthase regulates N-Ras activation on the Golgi complex of antigen-stimulated T cells
Proceedings of the National Academy of Sciences of the United States of America, Vol. 105, Núm. 30, pp. 10507-10512
2006
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Complex I dysfunction and tolerance to nitroglycerin: An approach based on mitochondrial-targeted antioxidants
Circulation Research, Vol. 99, Núm. 10, pp. 1067-1075
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Endothelial Nitric Oxide Synthase Regulates T Cell Receptor Signaling at the Immunological Synapse
Immunity, Vol. 24, Núm. 6, pp. 753-765
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Loss of apoptosis-inducing factor leads to an increase in reactive oxygen species, and an impairment of respiration that can be reversed by antioxidants [4]
Cell Death and Differentiation
2005
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Discrepancies between nitroglycerin and NO-releasing drugs on mitochondrial oxygen consumption, vasoactivity, and the release of NO
Circulation Research, Vol. 97, Núm. 10, pp. 1063-1069
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Role of free radicals in sepsis: Antioxidant therapy
Current Pharmaceutical Design, Vol. 11, Núm. 24, pp. 3141-3158