Publicaciones en las que colabora con M ELENA OBRADOR PLA (15)

2013

  1. Stress hormones promote growth of B16-F10 melanoma metastases: An interleukin 6- and glutathione-dependent mechanism

    Journal of Translational Medicine, Vol. 11, Núm. 1

2012

  1. Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1

    Journal of Translational Medicine, Vol. 10, Núm. 1

2011

  1. Intertissue flow of glutathione (GSH) as a tumor growth-promoting mechanism: Interleukin 6 induces GSH release from hepatocytes in metastatic B16 melanoma-bearing mice

    Journal of Biological Chemistry, Vol. 286, Núm. 18, pp. 15716-15727

2008

  1. Natural polyphenols facilitate elimination of HT-29 colorectal cancer xenografts by chemoradiotherapy: A Bcl-2- and superoxide dismutase 2-dependent mechanism

    Molecular Cancer Therapeutics, Vol. 7, Núm. 10, pp. 3330-3342

2007

  1. Bcl-2 and glutathione depletion sensitizes B16 melanoma to combination therapy and eliminates metastatic disease

    Clinical Cancer Research, Vol. 13, Núm. 9, pp. 2658-2666

  2. Nitric oxide mediates natural polyphenol-induced Bcl-2 down-regulation and activation of cell death in metastatic B16 melanoma

    Journal of Biological Chemistry, Vol. 282, Núm. 5, pp. 2880-2890

2006

  1. Bcl-2 and Mn-SOD antisense oligodeoxynucleotides and a glutamine-enriched diet facilitate elimination of highly resistant B16 melanoma cells by tumor necrosis factor-α and chemotherapy

    Journal of Biological Chemistry, Vol. 281, Núm. 1, pp. 69-79

2005

  1. Acceleration of glutathione efflux and inhibition of γ- glutamyltranspeptidase sensitize metastatic B16 melanoma cells to endothelium-induced cytotoxicity

    Journal of Biological Chemistry, Vol. 280, Núm. 8, pp. 6950-6959

  2. Association between pterostilbene and quercetin inhibits metastatic activity of B16 melanoma

    Neoplasia, Vol. 7, Núm. 1, pp. 37-47

2003

  1. Down-regulation of glutathione and Bcl-2 synthesis in mouse B16 melanoma cells avoids their survival during interaction with the vascular endothelium

    Journal of Biological Chemistry, Vol. 278, Núm. 41, pp. 39591-39599

  2. Tumor cytotoxicity by endothelial cells: Impairment of the mitochondrial system for glutathione uptake in mouse B16 melanoma cells that survive after in vitro interaction with the hepatic sinusoidal endothelium

    Journal of Biological Chemistry, Vol. 278, Núm. 16, pp. 13888-13897

2002

  1. Inhibition of cancer growth by resveratrol is related to its low bioavailability

    Free Radical Biology and Medicine, Vol. 33, Núm. 3, pp. 387-398

1998

  1. Regulation of tumour cell sensitivity to TNF-induced oxidative stress and cytotoxicity: Role of glutathione

    BioFactors

1997

  1. Blood glutathione as an index of radiation-induced oxidative stress in mice and humans

    Free Radical Biology and Medicine, Vol. 22, Núm. 7, pp. 1203-1209

  2. Glutathione and the rate of cellular proliferation determine tumour cell sensitivity to tumour necrosis factor in vivo

    Biochemical Journal, Vol. 325, Núm. 1, pp. 183-189