JOSE M
CENTENO GUIL
TITULAR DE UNIVERSIDAD
Publicaciones (36) Publicaciones de JOSE M CENTENO GUIL
2024
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Implantación de los objetivos de desarrollo sostenible en la Facultat de Farmàcia de la Universitat de València
Pharmaceutical care España, Vol. 26, Núm. 2, pp. 320-320
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Internacionalización de los estudiantes del grado en farmacia de la Universitat de València.
Pharmaceutical care España, Vol. 26, Núm. 2, pp. 327-327
2023
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Cerebroprotective Effect of 17β-Estradiol Replacement Therapy in Ovariectomy-Induced Post-Menopausal Rats Subjected to Ischemic Stroke: Role of MAPK/ERK1/2 Pathway and PI3K-Independent Akt Activation
International Journal of Molecular Sciences, Vol. 24, Núm. 18
2021
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The Role of NF-κB Triggered Inflammation in Cerebral Ischemia
Frontiers in Cellular Neuroscience, Vol. 15
2019
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Potassium channels contribute to the increased sensitivity of the rabbit carotid artery to hydrogen sulfide in diabetes
European Journal of Pharmacology, Vol. 853, pp. 33-40
2018
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Diabetes modifies the role of prostanoids and potassium channels which regulate the hypereactivity of the rabbit renal artery to BNP
Naunyn-Schmiedeberg's Archives of Pharmacology, Vol. 391, Núm. 5, pp. 501-511
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Molecular mechanisms underlying the neuroprotective role of atrial natriuretic peptide in experimental acute ischemic stroke
Molecular and Cellular Endocrinology, Vol. 472, pp. 1-9
2017
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Mechanisms involved in the increased sensitivity of the rabbit basilar artery to atrial natriuretic peptide in diabetes
European Journal of Pharmacology, Vol. 815, pp. 64-72
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Molecular mechanisms mediating the neuroprotective role of the selective estrogen receptor modulator, bazedoxifene, in acute ischemic stroke: A comparative study with 17β-estradiol
Journal of Steroid Biochemistry and Molecular Biology, Vol. 171, pp. 296-304
2014
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The selective estrogen receptor modulator, bazedoxifene, reduces ischemic brain damage in male rat
Neuroscience Letters, Vol. 575, pp. 53-57
2013
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Involvement of prostacyclin and potassium channels in the diabetes-induced hyporeactivity of the rabbit carotid artery to B-type natriuretic peptide
European Journal of Pharmacology, Vol. 701, Núm. 1-3, pp. 159-167
2012
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Diabetes impairs the atrial natriuretic peptide relaxant action mediated by potassium channels and prostacyclin in the rabbit renal artery
Pharmacological Research, Vol. 66, Núm. 5, pp. 392-400
2011
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Mechanisms underlying the diabetes-induced hyporeactivity of the rabbit carotid artery to atrial natriuretic peptide
Pharmacological Research, Vol. 63, Núm. 3, pp. 190-198
2010
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Mechanisms involved in the relaxant action of testosterone in the renal artery from male normoglycemic and diabetic rabbits
Pharmacological Research, Vol. 61, Núm. 2, pp. 149-156
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Role of NO-synthases and cyclooxygenases in the hyperreactivity of male rabbit carotid artery to testosterone under experimental diabetes
Pharmacological Research, Vol. 61, Núm. 1, pp. 62-70
2006
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Lactoferricin-related peptides with inhibitory effects on ACE-dependent vasoconstriction
Journal of Agricultural and Food Chemistry, Vol. 54, Núm. 15, pp. 5323-5329
2004
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Mechanisms underlying diabetes enhancement of endothelin-1-induced contraction in rabbit basilar artery
European Journal of Pharmacology, Vol. 486, Núm. 3, pp. 289-296
2002
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Acute relaxant effects of 17-β-estradiol through non-genomic mechanisms in rabbit carotid artery
Steroids, Vol. 67, Núm. 5, pp. 339-346
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Experimental diabetes induces hyperreactivity of rabbit renal artery to 5-hydroxytryptamine
European Journal of Pharmacology, Vol. 439, Núm. 1-3, pp. 121-127
2001
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Diabetes potentiates acetylcholine-induced relaxation in rabbit renal arteries
European Journal of Pharmacology, Vol. 415, Núm. 2-3, pp. 225-232