Publicacions en què col·labora amb Sonia Sánchez Campos (11)

2024

  1. Idiosyncratic Drug-Induced Liver Injury and Amoxicillin–Clavulanate: Spotlight on Gut Microbiota, Fecal Metabolome and Bile Acid Profile in Patients

    International Journal of Molecular Sciences, Vol. 25, Núm. 13

2021

  1. Molecular mechanisms of hepatotoxic cholestasis by clavulanic acid: Role of NRF2 and FXR pathways

    Food and Chemical Toxicology, Vol. 158

  2. The synbiotic combination of akkermansia muciniphila and quercetin ameliorates early obesity and NAFLD through gut microbiota reshaping and bile acid metabolism modulation

    Antioxidants, Vol. 10, Núm. 12

2019

  1. A Network Involving Gut Microbiota, Circulating Bile Acids, and Hepatic Metabolism Genes That Protects Against Non-Alcoholic Fatty Liver Disease

    Molecular Nutrition and Food Research, Vol. 63, Núm. 20

  2. Functional Interactions between Gut Microbiota Transplantation, Quercetin, and High-Fat Diet Determine Non-Alcoholic Fatty Liver Disease Development in Germ-Free Mice

    Molecular Nutrition and Food Research, Vol. 63, Núm. 8

2017

  1. Protective effect of quercetin on high-fat diet-induced non-alcoholic fatty liver disease in mice is mediated by modulating intestinal microbiota imbalance and related gut-liver axis activation

    Free Radical Biology and Medicine, Vol. 102, pp. 188-202

2016

  1. Hepatocyte vitamin D receptor regulates lipid metabolism and mediates experimental diet-induced steatosis

    Journal of Hepatology, Vol. 65, Núm. 4, pp. 748-757

2015

  1. Quercetin ameliorates dysregulation of lipid metabolism genes via the PI3K/AKT pathway in a diet-induced mouse model of nonalcoholic fatty liver disease

    Molecular Nutrition and Food Research, Vol. 59, Núm. 5, pp. 879-893

  2. Repression of the nuclear receptor small heterodimer partner by steatotic drugs and in advanced nonalcoholic fatty liver disease

    Molecular Pharmacology, Vol. 87, Núm. 4, pp. 582-594

2014

  1. Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin

    Laboratory Investigation, Vol. 94, Núm. 3, pp. 262-274

2013

  1. The human liver fatty acid binding protein (FABP1) gene is activated by FOXA1 and PPARα; And repressed by C/EBPα: Implications in FABP1 down-regulation in nonalcoholic fatty liver disease

    Biochimica et Biophysica Acta - Molecular and Cell Biology of Lipids, Vol. 1831, Núm. 4, pp. 803-818