JOSE M CENTENO GUIL-rekin lankidetzan egindako argitalpenak (8)

2018

  1. Diabetes modifies the role of prostanoids and potassium channels which regulate the hypereactivity of the rabbit renal artery to BNP

    Naunyn-Schmiedeberg's Archives of Pharmacology, Vol. 391, Núm. 5, pp. 501-511

2017

  1. Mechanisms involved in the increased sensitivity of the rabbit basilar artery to atrial natriuretic peptide in diabetes

    European Journal of Pharmacology, Vol. 815, pp. 64-72

2013

  1. Involvement of prostacyclin and potassium channels in the diabetes-induced hyporeactivity of the rabbit carotid artery to B-type natriuretic peptide

    European Journal of Pharmacology, Vol. 701, Núm. 1-3, pp. 159-167

2012

  1. Diabetes impairs the atrial natriuretic peptide relaxant action mediated by potassium channels and prostacyclin in the rabbit renal artery

    Pharmacological Research, Vol. 66, Núm. 5, pp. 392-400

2011

  1. Mechanisms underlying the diabetes-induced hyporeactivity of the rabbit carotid artery to atrial natriuretic peptide

    Pharmacological Research, Vol. 63, Núm. 3, pp. 190-198

2010

  1. Mechanisms involved in the relaxant action of testosterone in the renal artery from male normoglycemic and diabetic rabbits

    Pharmacological Research, Vol. 61, Núm. 2, pp. 149-156

  2. Role of NO-synthases and cyclooxygenases in the hyperreactivity of male rabbit carotid artery to testosterone under experimental diabetes

    Pharmacological Research, Vol. 61, Núm. 1, pp. 62-70

2004

  1. Mechanisms underlying diabetes enhancement of endothelin-1-induced contraction in rabbit basilar artery

    European Journal of Pharmacology, Vol. 486, Núm. 3, pp. 289-296