Publicaciones en las que colabora con M ELENA OBRADOR PLA (15)

2008

  1. Natural polyphenols facilitate elimination of HT-29 colorectal cancer xenografts by chemoradiotherapy: A Bcl-2- and superoxide dismutase 2-dependent mechanism

    Molecular Cancer Therapeutics, Vol. 7, Núm. 10, pp. 3330-3342

  2. Tumoricidal activity of endothelium-derived NO and the survival of metastatic cells with high GSH and Bcl-2 levels

    Nitric Oxide - Biology and Chemistry, Vol. 19, Núm. 2, pp. 107-114

2007

  1. Bcl-2 and glutathione depletion sensitizes B16 melanoma to combination therapy and eliminates metastatic disease

    Clinical Cancer Research, Vol. 13, Núm. 9, pp. 2658-2666

2006

  1. Bcl-2 and Mn-SOD antisense oligodeoxynucleotides and a glutamine-enriched diet facilitate elimination of highly resistant B16 melanoma cells by tumor necrosis factor-α and chemotherapy

    Journal of Biological Chemistry, Vol. 281, Núm. 1, pp. 69-79

2005

  1. Acceleration of glutathione efflux and inhibition of γ- glutamyltranspeptidase sensitize metastatic B16 melanoma cells to endothelium-induced cytotoxicity

    Journal of Biological Chemistry, Vol. 280, Núm. 8, pp. 6950-6959

2003

  1. Down-regulation of glutathione and Bcl-2 synthesis in mouse B16 melanoma cells avoids their survival during interaction with the vascular endothelium

    Journal of Biological Chemistry, Vol. 278, Núm. 41, pp. 39591-39599

  2. Tumor cytotoxicity by endothelial cells: Impairment of the mitochondrial system for glutathione uptake in mouse B16 melanoma cells that survive after in vitro interaction with the hepatic sinusoidal endothelium

    Journal of Biological Chemistry, Vol. 278, Núm. 16, pp. 13888-13897

2002

  1. Inhibition of cancer growth by resveratrol is related to its low bioavailability

    Free Radical Biology and Medicine, Vol. 33, Núm. 3, pp. 387-398

  2. γ-glutamyl transpeptidase overexpression increases metastatic growth of B16 melanoma cells in the mouse liver

    Hepatology, Vol. 35, Núm. 1, pp. 74-81

2001

  1. Glutamine potentiates TNF-α-induced tumor cytotoxicity

    Free Radical Biology and Medicine, Vol. 31, Núm. 5, pp. 642-650

  2. Possible mechanisms for tumour cell sensitivity to TNF-α and potential therapeutic applications

    Current Pharmaceutical Biotechnology, Vol. 2, Núm. 2, pp. 119-130

  3. Tumoricidal activity of endothelial cells: Inhibition of endothelial nitric oxide production abrogates tumor cytotoxicity induced by hepatic sinusoidal endothelium in response to B16 melanoma adhesion in vitro

    Journal of Biological Chemistry, Vol. 276, Núm. 28, pp. 25775-25782

2000

  1. Mitochondrial glutathione depletion by glutamine in growing tumor cells

    Free Radical Biology and Medicine, Vol. 29, Núm. 9, pp. 913-923

1999

  1. Changes in glutathione status and the antioxidant system in blood and in cancer cells associate with tumour growth in vivo

    Free Radical Biology and Medicine, Vol. 26, Núm. 3-4, pp. 410-418

  2. Growth-associated changes in glutathione content correlate with liver metastatic activity of B16 melanoma cells

    Clinical and Experimental Metastasis, Vol. 17, Núm. 7, pp. 567-574