Publicaciones en las que colabora con JOSE VIÑA RIBES (44)

2024

  1. Redox-associated changes in healthy individuals at risk of Alzheimer's disease. A ten-year follow-up study

    Free Radical Biology and Medicine, Vol. 215, pp. 56-63

2022

  1. Adult Neural Stem Cell Migration Is Impaired in a Mouse Model of Alzheimer’s Disease

    Molecular Neurobiology, Vol. 59, Núm. 2, pp. 1168-1182

2021

  1. Multicomponent Training Prevents Memory Deficit Related to Amyloid-β Protein-Induced Neurotoxicity

    Journal of Alzheimer's Disease, Vol. 83, Núm. 1, pp. 143-154

  2. Serum levels of clusterin, PKR, and RAGE correlate with amyloid burden in Alzheimer's disease

    Journal of Alzheimer's Disease, Vol. 80, Núm. 3, pp. 1067-1077

2020

  1. Increased basal antioxidant levels in RCAN1–deficient mice lowers oxidative injury after acute paraquat insult

    Free Radical Research, Vol. 54, Núm. 6, pp. 442-454

2019

  1. Oral Monosodium Glutamate Administration Causes Early Onset of Alzheimer's Disease-Like Pathophysiology in APP/PS1 Mice

    Journal of Alzheimer's Disease, Vol. 72, Núm. 3, pp. 957-975

2017

  1. New functions of APC/C ubiquitin ligase in the nervous system and its role in alzheimer’s disease

    International Journal of Molecular Sciences, Vol. 18, Núm. 5

2016

  1. Autoantibodies profile in matching CSF and serum from AD and AMCI patients: Potential pathogenic role and link to oxidative damage

    Current Alzheimer Research, Vol. 13, Núm. 2, pp. 112-122

  2. Aβ Induces Excitotoxicity Mediated by APC/C-Cdh1 Depletion That Can Be Prevented by Glutaminase Inhibition Promoting Neuronal Survival

    Scientific Reports, Vol. 6

  3. Oxidative signature of cerebrospinal fluid from mild cognitive impairment and Alzheimer disease patients

    Free Radical Biology and Medicine, Vol. 91, pp. 1-9

  4. Reductive stress: A new concept in alzheimer’s disease

    Current Alzheimer Research, Vol. 13, Núm. 2, pp. 206-211

2015

  1. Molecular mechanisms linking amyloid β toxicity and Tau hyperphosphorylation in Alzheimers disease

    Free Radical Biology and Medicine, Vol. 83, pp. 186-191

2014

  1. Aβ and tau toxicities in Alzheimer's are linked via oxidative stress-induced p38 activation: Protective role of vitamin E

    Redox Biology, Vol. 2, Núm. 1, pp. 873-877

2013

  1. Lymphocytes from young healthy persons carrying the ApoE4 Allele overexpress stress-related proteins involved in the pathophysiology of Alzheimer's disease

    Journal of Alzheimer's Disease, Vol. 33, Núm. 1, pp. 77-83

  2. Reductive stress in young healthy individuals at risk of Alzheimer disease

    Free Radical Biology and Medicine, Vol. 63, pp. 274-279

2011

  1. Amyloid-β toxicity and tau hyperphosphorylation are linked via RCAN1 in Alzheimer's disease

    Journal of Alzheimer's Disease, Vol. 27, Núm. 4, pp. 701-709

  2. Antioxidant pathways in Alzheimer's disease: Possibilities of intervention

    Current Pharmaceutical Design, Vol. 17, Núm. 35, pp. 3861-3864

2010

  1. Estradiol or genistein prevent Alzheimer's disease-associated inflammation correlating with an increase PPARγ expression in cultured astrocytes

    Brain Research, Vol. 1312, pp. 138-144

  2. Why women have more Alzheimer's disease than men: Gender and mitochondrial toxicity of amyloid-β peptide

    Journal of Alzheimer's Disease, Vol. 20, Núm. SUPPL.2

2009

  1. Vitamin e paradox in alzheimer's disease: It does not prevent loss of cognition and may even be detrimental

    Journal of Alzheimer's Disease, Vol. 17, Núm. 1, pp. 143-149