Publicaciones en las que colabora con Lucas Taoro González (19)

2022

  1. Hyperammonemia Alters the Function of AMPA and NMDA Receptors in Hippocampus: Extracellular cGMP Reverses Some of These Alterations

    Neurochemical Research, Vol. 47, Núm. 7, pp. 2016-2031

  2. Intracellular and extracelluar cyclic GMP in the brain and the hippocampus

    Vitamins and Hormones (Academic Press Inc.), pp. 247-288

2020

  1. Chronic hyperammonemia induces peripheral inflammation that leads to cognitive impairment in rats: Reversed by anti-TNF-α treatment

    Journal of Hepatology, Vol. 73, Núm. 3, pp. 582-592

2019

  1. Chronic hyperammonemia alters extracellular glutamate, glutamine and GABA and membrane expression of their transporters in rat cerebellum. Modulation by extracellular cGMP

    Neuropharmacology, Vol. 161

  2. Differential role of interleukin-1β in neuroinflammation-induced impairment of spatial and nonspatial memory in hyperammonemic rats

    FASEB Journal, Vol. 33, Núm. 9, pp. 9913-9928

  3. Extracellular cGMP Reverses Altered Membrane Expression of AMPA Receptors in Hippocampus of Hyperammonemic Rats: Underlying Mechanisms

    Molecular Neurobiology, Vol. 56, Núm. 6, pp. 4428-4439

  4. Peripheral inflammation induces neuroinflammation that alters neurotransmission and cognitive and motor function in hepatic encephalopathy: Underlying mechanisms and therapeutic implications

    Acta Physiologica, Vol. 226, Núm. 2

2018

  1. Chronic hyperammonemia alters in opposite ways membrane expression of GluA1 and GluA2 AMPA receptor subunits in cerebellum. Molecular mechanisms involved

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 1, pp. 286-295

  2. Hyperammonemia alters membrane expression of GluA1 and GluA2 subunits of AMPA receptors in hippocampus by enhancing activation of the IL-1 receptor: Underlying mechanisms

    Journal of Neuroinflammation, Vol. 15, Núm. 1

2017

  1. Extracellular Cyclic GMP Modulates Membrane Expression of the GluA1 and GluA2 Subunits of AMPA Receptor in Cerebellum: Molecular Mechanisms Involved

    Scientific Reports, Vol. 7, Núm. 1

  2. Sildenafil reduces neuroinflammation in cerebellum, restores GABAergic tone, and improves motor in-coordination in rats with hepatic encephalopathy

    CNS Neuroscience and Therapeutics, Vol. 23, Núm. 5, pp. 386-394

2016

  1. Extracellular cGMP Modulates Learning Biphasically by Modulating Glycine Receptors, CaMKII and Glutamate-Nitric Oxide-cGMP Pathway

    Scientific Reports, Vol. 6

  2. Hyperammonemia alters glycinergic neurotransmission and modulation of the glutamate-nitric oxide-cGMP pathway by extracellular glycine in cerebellum in vivo

    Journal of Neurochemistry, Vol. 137, Núm. 4, pp. 539-548

  3. Hyperammonemia induces glial activation, neuroinflammation and alters neurotransmitter receptors in hippocampus, impairing spatial learning: Reversal by sulforaphane

    Journal of Neuroinflammation, Vol. 13, Núm. 1

  4. In vivo administration of extracellular cGMP normalizes TNF-α and membrane expression of AMPA receptors in hippocampus and spatial reference memory but not IL-1β, NMDA receptors in membrane and working memory in hyperammonemic rats

    Brain, Behavior, and Immunity, Vol. 57, pp. 360-370

  5. Infliximab reduces peripheral inflammation, neuroinflammation, and extracellular GABA in the cerebellum and improves learning and motor coordination in rats with hepatic encephalopathy

    Journal of Neuroinflammation, Vol. 13, Núm. 1

  6. Neuroinflammation increases GABAergic tone and impairs cognitive and motor function in hyperammonemia by increasing GAT-3 membrane expression. Reversal by sulforaphane by promoting M2 polarization of microglia

    Journal of Neuroinflammation, Vol. 13, Núm. 1

  7. Reducing peripheral inflammation with infliximab reduces neuroinflammation and improves cognition in rats with hepatic encephalopathy

    Frontiers in Molecular Neuroscience, Vol. 9, Núm. NOV2016

2015

  1. Sildenafil reduces neuroinflammation and restores spatial learning in rats with hepatic encephalopathy: Underlying mechanisms

    Journal of Neuroinflammation, Vol. 12, Núm. 1