BELEN MOLLA MOLINER-rekin lankidetzan egindako argitalpenak (7)

2022

  1. Frataxin Deficit Leads to Reduced Dynamics of Growth Cones in Dorsal Root Ganglia Neurons of Friedreich’s Ataxia YG8sR Model: A Multilinear Algebra Approach

    Frontiers in Molecular Neuroscience, Vol. 15

2020

  1. Cofilin dysregulation alters actin turnover in frataxin-deficient neurons

    Scientific Reports, Vol. 10, Núm. 1

2019

  1. Phosphodiesterase Inhibitors Revert Axonal Dystrophy in Friedreich’s Ataxia Mouse Model

    Neurotherapeutics, Vol. 16, Núm. 2, pp. 432-449

2017

  1. Reversible axonal dystrophy by calcium modulation in frataxin-deficient sensory neurons of YG8R mice

    Frontiers in Molecular Neuroscience, Vol. 10

2016

  1. Two different pathogenic mechanisms, dying-back axonal neuropathy and pancreatic senescence, are present in the YG8R mouse model of Friedreich's ataxia

    DMM Disease Models and Mechanisms, Vol. 9, Núm. 6, pp. 647-657

2014

  1. Mitochondrial dysfunction induced by frataxin deficiency is associated with cellular senescence and abnormal calcium metabolism

    Frontiers in Cellular Neuroscience, Vol. 8, Núm. MAY

2011

  1. Differential expression of PGC-1α and metabolic sensors suggest age-dependent induction of mitochondrial biogenesis in friedreich ataxia fibroblasts

    PLoS ONE, Vol. 6, Núm. 6